Which is Preferable for Hair Loss - RU58841 or Finasteride?

Hair loss is one of the most unpleasant side effects of AAS use. Most people don't want to lose their hair for the sake of greater muscle mass, so it's important to know how to detect hair loss in its early stages and how to possibly stop it.

Happily, not all people are susceptible to hair loss from AAS use, and not all AAS cause hair loss. Nevertheless, it is useful to know what treatments can help treat hair loss and how to use them with greater result.

In this article, we will learn the causes of hair loss caused by some AAS and compare two popular hair loss medications: finasteride and RU-58841.

Dihydrotestosterone (DHT) is a 5-alpha-reduced form of testosterone, which is associated with male virilization, sexual development, sexual desire, sexual function and mood.

While DHT is great and necessary for us to perform at our best, it can cause hair loss by causing hair follicles to shrink (a phenomenon called androgenic alopecia) in men who have a genetic predisposition to it. Certain AAS can cause the same side effects by "acting" as DHT in the body.

Some examples of AAS that may cause alopecia in men susceptible to the condition include Testosterone, Winstrol, Masteron, Proviron and Primobolan among others.

As we discussed how and why hair loss happens, here's an overview of the major hair loss remedies and how their effectiveness compares....

Finasteride is a commonly prescribed drug for preventing hair loss. It is an oral steroidal anti-androgen that reduces DHT levels by inhibiting the 5-alpha-reductase enzyme, which is responsible for converting testosterone into DHT.

By decreasing circulating DHT levels, it can help reduce the occurrence of androgenic alopecia and acne, allowing men to maintain their hair and smooth skin. Unfortunately, a reduction in DHT can cause erectile dysfunction, loss of libido, and depression in a significant number of users.

RU-58841 is a non-steroidal anti-androgen that is exclusively used topically. It binds to androgen receptors wherever it is applied, preventing DHT from attaching to them, instead of inhibiting the conversion of testosterone into DHT.